Non human primates and human clinical sample support:
Quantitate the number and regional occupancy of protein aggregates in transgenic mouse models of neurodegenerative disease after perturbing cell signaling pathways.
Measure the effectiveness of therapeutic interventions on beta amyloid plaque load for treatment of Alzheimer’s Disease.
Study the biology of protein aggregate formation and spread across neural circuits throughout the brain.
For additional publications supporting this use case refer to:
Hoglund et al. (2024) Actaneuropathologica
Jiang et al. (2024) Frontiers in Neuroscience
Chen et al. (2023) PNAS
Soula et al. (2023) Nature Neuroscience
Martorell et al. (2019) Cell
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